
Semax (C-MAX) 10 mg
$150.00
Quantity
A synthetic ACTH‑derived heptapeptide studied for neuroprotective and cognitive effects.
No. Semax (C‑Max) is not naturally occurring — it’s a synthetic heptapeptide, an analog of the adrenocorticotropic hormone (ACTH) fragment.
Studies on ischemia, neuroplasticity, BDNF regulation, and cognitive function.
Keep refrigerated at 2–8 °C for short‑term storage; −20 °C for long‑term. Avoid repeated freeze‑thaw cycles.
24 months in lyophilized form; 2–4 weeks after reconstitution if refrigerated.
No, it is strictly for research purposes.
C‑Max (Semax) is a synthetic heptapeptide derived from the adrenocorticotropic hormone (ACTH) fragment. It is classified as a neuroactive peptide and has been studied for its potential roles in neuroprotection, cognitive enhancement, and regulation of brain‑derived neurotrophic factor (BDNF).
Origin: Developed in Russia in the late 1980s as a therapeutic analog of ACTH fragments.
Research Focus: Initially investigated for ischemic stroke recovery and cognitive disorders.
Modern Studies: Expanded into areas of neuroplasticity, learning, memory, and stress resilience.
Neuroprotection: Demonstrated protective effects against ischemic brain injury in animal models.
Cognitive Function: Enhances learning and memory performance in preclinical studies.
BDNF Regulation: Shown to upregulate brain‑derived neurotrophic factor, supporting synaptic plasticity.
Stress Response: Modulates neurotransmitter systems linked to anxiety and adaptation.

Ashmarin IP, et al. Semax: A New Peptide Drug for Treatment of Stroke. Neurosci Behav Physiol.
Skorik YA, et al. Semax and Neurotrophic Factors. Neurochem J.
PubChem CID 250744 — Semax peptide profile.



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